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Healthy Lifestyle

Improvements in Nutrition and Lifestyle Increase Telomerase Activity

Introduction Telomeres, the protective DNA–protein complexes at the end of chromosomes, are required for DNA replication and to protect chromosomes from nuclease degradation, end-to-end fusion, and the initiation of cellular senescence. Since telomeres shorten with each cell division, telomere length is a key indicator of mitotic cell aging and viability. Telomere length has emerged as […]

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y-Tocopherol, but Not a-Tocopherol, Improves Anti-Inflammatory; Decreases Adverse Effects of Aspirins

Nonsteriodal anti-inflammatory drugs (NSAIDs), including aspirin, are commonly used for pain relief, have consistently been shown to reduce the risk of cancers, and low-dose aspirin is effective in reducing cardiovascular disease risk because of its antithrombotic effect. However, the long-term use of aspirin and other NSAIDs has been hindered by their associated adverse effects, including […]

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The UltraMind Solution: A Functional Medicine Approach to Brain Disorders

Functional Medicine: A 21st Century Systems Biology Approach to Individualized Clinical Care. Dr. Mark Hyman’s latest book, The UltraMind Solution (Scribner’s, 2009) presents a highly convincing argument, backed by hundreds of peer-reviewed studies published within the last five years, for the validity of a functional medicine approach to treating brain disorders of all types. Functional […]

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Chromosomes

Slowing Telomere Attrition and Cellular Senescence—Today: Part II

Abstract Cellular senescence, although protective when triggered in cells that have become cancerous, plays a key pathological role in other age-associated diseases and in so-called “healthy aging.” The age-associated increase in cellular senescence has been assumed to result from the cumulative effects of “wear and tear,” but genetic research has revealed that cellular senescence occurs […]

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Chromosomes

Slowing Telomere Attrition and Cellular Senescence—Today: Part I

Abstract Cellular senescence, although protective when triggered in cells that have become cancerous, plays a key pathological role in other age-associated diseases and in so-called “healthy aging.” The age-associated increase in cellular senescence has been assumed to result from the cumulative effects of “wear and tear,” but genetic research has revealed that cellular senescence occurs […]

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